In the last years, collaborative studies have joined to link the degree of genetic heterogeneity of acute myeloid leukemia (AML) to clinical outcome,1, 2 allowing risk stratification before therapy and guiding post-induction treatment of children with AML. So far, still half of these patients, whose disease is usually characterized by a grim prognosis, lack a known biomarker offering opportunities of targeted treatment.
NUP98-fusion transcripts characterize different biological entities within acute myeloid leukemia: A report from the AIEOP-AML group / Bisio, V.; Zampini, M.; Tregnago, C.; Manara, E.; Salsi, Valentina; Di Meglio, A.; Masetti, R.; Togni, M.; Di Giacomo, D.; Minuzzo, S.; Leszl, A.; Zappavigna, Vincenzo; Rondelli, R.; Mecucci, C.; Pession, A.; Locatelli, F.; Basso, G.; Pigazzi, M.. - In: LEUKEMIA. - ISSN 0887-6924. - 31:4(2017), pp. 974-977. [10.1038/leu.2016.361]
NUP98-fusion transcripts characterize different biological entities within acute myeloid leukemia: A report from the AIEOP-AML group
SALSI, Valentina;ZAPPAVIGNA, Vincenzo;
2017
Abstract
In the last years, collaborative studies have joined to link the degree of genetic heterogeneity of acute myeloid leukemia (AML) to clinical outcome,1, 2 allowing risk stratification before therapy and guiding post-induction treatment of children with AML. So far, still half of these patients, whose disease is usually characterized by a grim prognosis, lack a known biomarker offering opportunities of targeted treatment.
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