In the last years, collaborative studies have joined to link the degree of genetic heterogeneity of acute myeloid leukemia (AML) to clinical outcome,1, 2 allowing risk stratification before therapy and guiding post-induction treatment of children with AML. So far, still half of these patients, whose disease is usually characterized by a grim prognosis, lack a known biomarker offering opportunities of targeted treatment.
NUP98-fusion transcripts characterize different biological entities within acute myeloid leukemia: A report from the AIEOP-AML group / Bisio, V., Zampini, M., Tregnago, C., Manara, E., Salsi, V., Di Meglio, A., Masetti, R., Togni, M., Di Giacomo, D., Minuzzo, S., Leszl, A., Zappavigna, V., Rondelli, R., Mecucci, C., Pession, A., Locatelli, F., Basso, G., Pigazzi, M.. - In: LEUKEMIA. - ISSN 0887-6924. - 31:4(2017), pp. 974-977. [10.1038/leu.2016.361]
NUP98-fusion transcripts characterize different biological entities within acute myeloid leukemia: A report from the AIEOP-AML group
SALSI, Valentina;ZAPPAVIGNA, Vincenzo;
2017
Abstract
In the last years, collaborative studies have joined to link the degree of genetic heterogeneity of acute myeloid leukemia (AML) to clinical outcome,1, 2 allowing risk stratification before therapy and guiding post-induction treatment of children with AML. So far, still half of these patients, whose disease is usually characterized by a grim prognosis, lack a known biomarker offering opportunities of targeted treatment.
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