Reactivation of the varicella-zoster virus (VZV) along the sensory nerves innervating the ear, including the geniculate ganglion, is responsible for herpes zoster oticus (HZO). In some cases, HZO is associated with polyneuropathy of the cranial nerves, although the mechanism of this involvement is not known. To explain this phenomenon and based on some clinical considerations, the present authors hypothesize an intersynaptic spread of VZV along the reflex pathways of the brainstem
Herpes zoster oticus: a clinical model for a transynaptic, reflex pathways, viral transmission hypotheses / ALICANDRI CIUFELLI, Matteo; Genovese, Elisabetta; Monzani, Daniele; Presutti, Livio; Aggazzotti Cavazza, Elisa. - In: NEUROSCIENCE RESEARCH. - ISSN 0168-0102. - ELETTRONICO. - 74:1(2012), pp. 7-9. [10.1016/j.neures.2012.06.001]
Herpes zoster oticus: a clinical model for a transynaptic, reflex pathways, viral transmission hypotheses
ALICANDRI CIUFELLI, Matteo;GENOVESE, Elisabetta;MONZANI, Daniele;PRESUTTI, Livio;Aggazzotti Cavazza, Elisa
2012
Abstract
Reactivation of the varicella-zoster virus (VZV) along the sensory nerves innervating the ear, including the geniculate ganglion, is responsible for herpes zoster oticus (HZO). In some cases, HZO is associated with polyneuropathy of the cranial nerves, although the mechanism of this involvement is not known. To explain this phenomenon and based on some clinical considerations, the present authors hypothesize an intersynaptic spread of VZV along the reflex pathways of the brainstem
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