Inhibition of nitric oxide overproduction, directly detected in the blood by ESR spectrometry, is involved in the mechanism of action of ACTH-(1-24) in hemorrhagic shock reversal.
Inhibition of nitric oxide overproduction, directly detected in the blood by ESR spectrometry, is involved in the mechanism of action of ACTH-(1-24) in hemorrhagic shock reversal / Guarini, Salvatore; Bazzani, Carla; Bini, Anna; Tomasi, Aldo; Bertolini, Alfio. - In: LE BASI RAZIONALI DELLA TERAPIA. - STAMPA. - 27:(1997), pp. 55-55. (Intervento presentato al convegno 3° Incontro Nazionale di Farmacologia del Cuore dei Vasi e del Microcircolo tenutosi a Maratea nel 20-21 Settembre 1996).
Inhibition of nitric oxide overproduction, directly detected in the blood by ESR spectrometry, is involved in the mechanism of action of ACTH-(1-24) in hemorrhagic shock reversal.
GUARINI, Salvatore;BAZZANI, Carla;BINI, Anna;TOMASI, Aldo;BERTOLINI, Alfio
1997
Abstract
Inhibition of nitric oxide overproduction, directly detected in the blood by ESR spectrometry, is involved in the mechanism of action of ACTH-(1-24) in hemorrhagic shock reversal.
Pubblicazioni consigliate
I metadati presenti in IRIS UNIMORE sono rilasciati con licenza Creative Commons CC0 1.0 Universal, mentre i file delle pubblicazioni sono rilasciati con licenza Attribuzione 4.0 Internazionale (CC BY 4.0), salvo diversa indicazione.
In caso di violazione di copyright, contattare Supporto Iris
