There are at least two factors that play a key role in vulvovaginal candidiasis: the massive infiltration of neutrophils and extensive presence of hyphae in the vagina. Thus it is conceivable that the dynamic of inflammatory response starts with adherence of Candida to epithelial cells followed by release of S100A and inflammasome activation with IL-1β production and subsequent further increase of PMN migration and activation.
Experimental models of vaginal candidiasis and inflammation / Vecchiarelli, Anna; Gabrielli, Elena; Pericolini, Eva. - In: FUTURE MICROBIOLOGY. - ISSN 1746-0913. - 10:8(2015), pp. 1265-1268. [10.2217/FMB.15.52]
Experimental models of vaginal candidiasis and inflammation
PERICOLINI, Eva
2015
Abstract
There are at least two factors that play a key role in vulvovaginal candidiasis: the massive infiltration of neutrophils and extensive presence of hyphae in the vagina. Thus it is conceivable that the dynamic of inflammatory response starts with adherence of Candida to epithelial cells followed by release of S100A and inflammasome activation with IL-1β production and subsequent further increase of PMN migration and activation.
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