These observations suggest that encephalopathy due to fulminant hepatic faliure (FHF) may precipitate in the presence of low level of endogenous benzodiazepine and that these compounds may accumulate in the blood during the course of FHF without inducing encephalopath. One of the mechanisms of the increse of these compounds in the blood seems to be decreased urinary excretion due to diuresis.
Endogenous benzodiazepine-like compounds in a patients with fulminatn hepatic failure: influence of a decreased renal excretion / R., Cosenza; I., Venturini; L., Miglioli; Farina, Franco; Corsi, Lorenzo; Avallone, Rossella; R., Amedei; G., Ardizzone; A., Arrigo; E., Minelli; S., Stefanelli; Baraldi, Mario; Zeneroli, Maria Luisa. - STAMPA. - 29 suppl.2:(1997), pp. A181-A181. (Intervento presentato al convegno national Congress os Ddigestive disease tenutosi a Bologna nel 22-27 novembre 1997).
Endogenous benzodiazepine-like compounds in a patients with fulminatn hepatic failure: influence of a decreased renal excretion
FARINA, Franco;CORSI, Lorenzo;AVALLONE, Rossella;BARALDI, Mario;ZENEROLI, Maria Luisa
1997
Abstract
These observations suggest that encephalopathy due to fulminant hepatic faliure (FHF) may precipitate in the presence of low level of endogenous benzodiazepine and that these compounds may accumulate in the blood during the course of FHF without inducing encephalopath. One of the mechanisms of the increse of these compounds in the blood seems to be decreased urinary excretion due to diuresis.
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